Your candy can control this bowel bacterium
Mouse and human studies suggest regulating the intestinal microbial and hormone-glp-1 hunger
We hate acknowledging, but we are not complete control of our own limbs; Bacteria can sometimes be the highestA cup of cookie or a glass too. A recent study in Natural microbiology Identifies the link between the abundance of a common bacterium in a person’s bowel and the amount of the sugar consumed by a person. Results may help researchers develop new treatments for many metabolic conditions.
Yong Q. Chen, the University of China University Cancer Cancer, and his team was investigating the role of a protein in the process of Fatty acid free receptors (FFAR4) in the process of fat metabolism. At first the rodents placed in high fat. “One day, I suggested a diet for high carbohydrates (high sugar) for comparison, and the results were amazing,” said Chenek. “We expected a fatty acid that can regulate fat priority. Even if it is amazing, it modulates the longing of sugar instead.”
Chen’s team found that the FFAR4 less ffar4 was related to higher priority for high-sugar diets. Researchers also compare FFAR4 levels with Diabetes with diabetes with diabetes. That’s where the gut is included in the microbiome; Scientists also discovered the mouse, the abundance of a microbe called that it was linked to lower levels of FFAR4 Ordinary bacteria.
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Researchers investigated how this bacterium can participate and found a metabolite created B. VulgatePantotenate – It is known as vitaminJust-Tiggers hormone GLP-1 production, which regulates hunger. In other words, FFAR4 means less B. VulgateLess pantotive and less GLP-1.
This newly identified way to identify recipients, hormones and the desire to eat our gut microbes to keep healthy working with just a stealthy work.
“I was glad that the study supports the existence of the innocent interactions between the host and microbiose,” says Sergueï O. Fetissov, the Physius of the University of Normandy in the French Rouen, who did not participate in the study. Stated the identification of the pantotenate B. Vulgate As a molecule that stimulates the GLP-1 secretion and the priority of sugar is reduced “an important discovery” because it can open new treatment for type 2 diabetes.
Elisa Caffrey, a doctoral candidate for the University of Stanford University, agrees, highlights potential Vitamin BJust Additional or even a drug to increase the amount of FFAR4, even more research, including clinical trials, properly. (Caffrey didn’t participate in the study.)
But there are still unanswered questions. B. Vulgate It is not the only microbe that affects GLP-1 production; Fetissov’s team found before They showed chill It also stimulates release. Uniform B. Vulgate More exploration with LPG-1-1 factor regulators, Chen says.
